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Characterisation of the inflammatory cytokine and growth . . . To clarify the mechanisms and their temporal relationship in the development of proliferative vitreoretinopathy (PVR), we measured vitreous levels of pro-inflammatory cytokines and growth factors
Congressionally Directed Medical Research Programs (CDMRP . . . PUBLIC ABSTRACT The overall goal of this proposal is to address the lack of treatment for proliferative vitreoretinopathy (PVR), a major cause of severe and irreversible visual loss in ocular trauma patients The incidence of blindness from PVR is particularly high amongst active Service Members due to the combat-related risks of exposure to explosive devices, blast and projectiles
Determining the effect of low-dose isotretinoin on . . . InTroduCTIon Proliferative vitreoretinopathy (PVR) refers to the development of periretinal fibrocellular membranes in the setting of rhegmatogenous retinal detach-ment (RRD) and occurs in approximately 5% to 10% of all retinal detachment cases 1 This process is believed to be due to the transdifferentiation and migration of retinal pigment epithelial cells and associated glial cells through a
p21CIP WAF1 saRNA inhibits proliferative vitreoretinopathy in . . . Proliferative vitreoretinopathy (PVR) is a disease process resulting from proliferation of reti-nal pigment epithelial (RPE) cells in the vitreous and periretinal area, leading to periretinal membrane formation and traction and eventually to postoperative failure after vitreo-retinal surgery for primary rhegmatogenous retinal detachment (RRD)
PII: S0161-6420(00)00076-2 - Ophthalmology GRT giant retinal tear; PVR proliferative vitreoretinopathy; RD 5 5 5 retinal detachment; SD standard deviation 5 vitreoretinopathy than did the Vitreon group (P 0 001) Out-, come variables analyzed were, therefore, stratified by diagnostic category No difference was found between the two groups in the proportion of primary versus
Mechanisms in proliferative vitreoretinopathy The wound-healing paradigm for proliferative vitreoretinopathy has provided numerous avenues for investigation into the pathobiology of the process The emerging picture is a complex interaction of cy-tokines, matrix proteins, and metalloproteinases influ-encing cell behavior and resulting in the formation of undesirable periretinal membranes