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Immune dysfunction in Alzheimer disease - PubMed This Review examines peripheral immune alterations in AD, evaluates related therapeutic opportunities and highlights key knowledge gaps, particularly the need for human-derived data to advance translational progress
The role of peripheral innate immune cells in Alzheimer’s disease . . . While microglia and astrocytes dominate CNS immune responses, emerging evidence implicates peripheral innate immune cells (PIICs)—including neutrophils, monocytes, dendritic cells, NK cells, and myeloid-derived suppressor cells (MDSCs)—as critical modulators of AD pathogenesis
NLRP3 inflammasome and Alzheimer’s disease: bridging . . . - Springer The progressive neurodegenerative disease known as Alzheimer’s disease (AD) is characterized by widespread neuronal death, memory loss, and cognitive decline The NLRP3 inflammasome has emerged as a key modulator of neuroinflammation, which is increasingly implicated in the pathophysiology of AD In response to endogenous and pathogenic danger signals, the innate immune system’s
Immune dysfunction in Alzheimer disease - Nature Advances in neuroimmunology have revealed that immune cell crosstalk involving T cells, B cells, monocytes and or macrophages and neutrophils can modulate the hallmark features of AD, including
Alzheimers Dementia - Wiley Online Library The Alzheimer’s Association® advances research to end Alzheimer’s and dementia while enhancing care for those living with the diseases The Alzheimer’s Association is the largest non-profit funder of Alzheimer’s disease research